SHADES OF SHOCK 5

4.5 Decreased respiratory ventilation and SHOCK LUNG
There is a decrease in available O2 in these in all cases as OF:
i) Pneumothorax
ii) Pulmonary embolus
iii) Shock lung or adult respiratory distress syndrome (ARDS).
This is met with in trauma, sepsis, aspiration of
gastric contents, and pneumonia

All these conditions lead to injury to the lung
tissue resulting in damage to the alveolar epithelium/
Capillary bed interface producing leakage of protein
Rich fluid into the interstitial space and later into
The alveolar space leading to respiratory distress.
This impairs gas exchange capacity of the lung The clinical features of ARDS of hypoxia, low lung
compliance and all these conditions interfere with
tissue perfusion with O2 because of impaired gas
exchange.
A diffuse interstitial pattern on X-ray is
visible.

4.1.1+ 2 and 4.3 are due to Pre-load and after-load effects
while 4.4 is due to cardiac muscle fuction impairment (infarct
/compression). All have low available O2 in the
blood.
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